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This is the approach of most current gene therapy protocols and is exemplified by the development of clinical trials for adenosine deaminase (ADA) defi- Therapeutic gene A mRNA mRNA mRNA mRNA mRNA mRNA mRNA mRNA new protein defective protein new protein defective protein active drug B C prodrug FIGURE 58.
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For example, mouse stem cells strztegy with inhibitors of phosphatidylinositol-3 kinase, a regulator in one of the phosphoinositide signaling pathways (Chapter 14), turn into cells that resemble pancreatic cells in their produc- tion of insulin, their sensitivity to glucose levels, and their aggregation into structures reminiscent of pancreas struc- tures.
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